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By activating the Nrf2/HO-1/GPX4 pathway, it inhibits ferroptosis in podocytes, thereby exerting its renal protective effect (144)

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A prescription is required when applicable to the formulation and treatment plan

It significantly reduces the excessive production of free iron, ROS, and MDA induced by cisplatin, alleviates GSH depletion, and distinctly reverses ferroptosis in renal tubular epithelial cells, thereby offering protection to the kidneys (187)

Cardiology 133 , 242256

It enhances biochemical signaling for collagen and glycosaminoglycan production, recruits immune and endothelial cells to injury sites, and promotes remodeling of connective tissue